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These results indicated that a large number of effector cells accumulated in lung tissue after CD5/ LNP-FAPCAR 2.2 treatment because of T cell immune activation, which partially resulted from transient FAPCAR-T recruited in the fibrotic lung
Stimulates fibroblasts to produce type I and III collagen, glycosaminoglycans, and decorin rebuilding the extracellular matrix architecture in damaged tissue
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